wnt β catenin signaling agonist skl200134 (Selleck Chemicals)
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wnt β catenin signaling agonist skl200134
Wnt β Catenin Signaling Agonist Skl200134, supplied by Selleck Chemicals, used in various techniques. Bioz Stars score: 94/100, based on 81 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/wnt+agonist+skl2001/SKL2001/pm41398307-166-7-15
Average 94 stars, based on 81 article reviews
Wnt β Catenin Signaling Agonist Skl200134, supplied by Selleck Chemicals, used in various techniques. Bioz Stars score: 94/100, based on 81 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/wnt+agonist+skl2001/SKL2001/pm41398307-166-7-15
Average 94 stars, based on 81 article reviews
wnt β catenin signaling agonist skl200134 - by Bioz Stars,
2026-10
94/100 stars
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Activation Assay:Article Title: CD36 enrichment in HER2-positive mesenchymal stem cells drives therapy refractoriness in breast cancer. Article Snippet: To examine the contribution of FA uptake via CD36 in the formation of HER2 + EMT-like CSCs, HCC1954, MDAMB361, EFM192A and HCC1569 cells were plated and grown in a 6-well plate at a density of 150,000– 400,000 cells/well (according to the cell line) and incubated in culture media supplemented with FBS depleted (FBS-FA-) or not (FBS-FA +) of FA for 24 h, 48 h and 72 h. To evaluate the effects of selective WNT inhibition, HCC1954, MDAMB361, EFM192A and HCC1569 cells were treated with the WNT inhibitor LGK974 (Selleckchem, Karl-Schmid-Str.14, Munich, DE) as a single agent at final concentrations of 10 μM and 20 μM (HCC1954, MDAMB361 and EFM192A cells) or 15 μM and 30 μM (HCC1569 cells) or with 0.1% DMSO (Sigma‒Aldrich) as an internal control for 24 h and 48 h at 37 °C with 5% CO2. .. In parallel, to evaluate the effects of selective WNT activation, HCC1954, MDAMB361 and EFM192A cells were treated with the specific Article Title: CD36 enrichment in HER2-positive mesenchymal stem cells drives therapy refractoriness in breast cancer Article Snippet: To examine the contribution of FA uptake via CD36 in the formation of HER2 + EMT-like CSCs, HCC1954, MDAMB361, EFM192A and HCC1569 cells were plated and grown in a 6-well plate at a density of 150,000–400,000 cells/well (according to the cell line) and incubated in culture media supplemented with FBS depleted (FBS-FA-) or not (FBS-FA +) of FA for 24 h, 48 h and 72 h. To evaluate the effects of selective WNT inhibition, HCC1954, MDAMB361, EFM192A and HCC1569 cells were treated with the WNT inhibitor LGK974 (Selleckchem, Karl-Schmid-Str.14, Munich, DE) as a single agent at final concentrations of 10 μM and 20 μM (HCC1954, MDAMB361 and EFM192A cells) or 15 μM and 30 μM (HCC1569 cells) or with 0.1% DMSO (Sigma‒Aldrich) as an internal control for 24 h and 48 h at 37 °C with 5% CO 2 . .. In parallel, to evaluate the effects of selective WNT activation, HCC1954, MDAMB361 and EFM192A cells were treated with the specific Control:Article Title: CD36 enrichment in HER2-positive mesenchymal stem cells drives therapy refractoriness in breast cancer. Article Snippet: To examine the contribution of FA uptake via CD36 in the formation of HER2 + EMT-like CSCs, HCC1954, MDAMB361, EFM192A and HCC1569 cells were plated and grown in a 6-well plate at a density of 150,000– 400,000 cells/well (according to the cell line) and incubated in culture media supplemented with FBS depleted (FBS-FA-) or not (FBS-FA +) of FA for 24 h, 48 h and 72 h. To evaluate the effects of selective WNT inhibition, HCC1954, MDAMB361, EFM192A and HCC1569 cells were treated with the WNT inhibitor LGK974 (Selleckchem, Karl-Schmid-Str.14, Munich, DE) as a single agent at final concentrations of 10 μM and 20 μM (HCC1954, MDAMB361 and EFM192A cells) or 15 μM and 30 μM (HCC1569 cells) or with 0.1% DMSO (Sigma‒Aldrich) as an internal control for 24 h and 48 h at 37 °C with 5% CO2. .. In parallel, to evaluate the effects of selective WNT activation, HCC1954, MDAMB361 and EFM192A cells were treated with the specific Article Title: CD36 enrichment in HER2-positive mesenchymal stem cells drives therapy refractoriness in breast cancer Article Snippet: To examine the contribution of FA uptake via CD36 in the formation of HER2 + EMT-like CSCs, HCC1954, MDAMB361, EFM192A and HCC1569 cells were plated and grown in a 6-well plate at a density of 150,000–400,000 cells/well (according to the cell line) and incubated in culture media supplemented with FBS depleted (FBS-FA-) or not (FBS-FA +) of FA for 24 h, 48 h and 72 h. To evaluate the effects of selective WNT inhibition, HCC1954, MDAMB361, EFM192A and HCC1569 cells were treated with the WNT inhibitor LGK974 (Selleckchem, Karl-Schmid-Str.14, Munich, DE) as a single agent at final concentrations of 10 μM and 20 μM (HCC1954, MDAMB361 and EFM192A cells) or 15 μM and 30 μM (HCC1569 cells) or with 0.1% DMSO (Sigma‒Aldrich) as an internal control for 24 h and 48 h at 37 °C with 5% CO 2 . .. In parallel, to evaluate the effects of selective WNT activation, HCC1954, MDAMB361 and EFM192A cells were treated with the specific Expressing:Article Title: CD36 enrichment in HER2-positive mesenchymal stem cells drives therapy refractoriness in breast cancer. Article Snippet: To examine the contribution of FA uptake via CD36 in the formation of HER2 + EMT-like CSCs, HCC1954, MDAMB361, EFM192A and HCC1569 cells were plated and grown in a 6-well plate at a density of 150,000– 400,000 cells/well (according to the cell line) and incubated in culture media supplemented with FBS depleted (FBS-FA-) or not (FBS-FA +) of FA for 24 h, 48 h and 72 h. To evaluate the effects of selective WNT inhibition, HCC1954, MDAMB361, EFM192A and HCC1569 cells were treated with the WNT inhibitor LGK974 (Selleckchem, Karl-Schmid-Str.14, Munich, DE) as a single agent at final concentrations of 10 μM and 20 μM (HCC1954, MDAMB361 and EFM192A cells) or 15 μM and 30 μM (HCC1569 cells) or with 0.1% DMSO (Sigma‒Aldrich) as an internal control for 24 h and 48 h at 37 °C with 5% CO2. .. In parallel, to evaluate the effects of selective WNT activation, HCC1954, MDAMB361 and EFM192A cells were treated with the specific Article Title: CD36 enrichment in HER2-positive mesenchymal stem cells drives therapy refractoriness in breast cancer Article Snippet: To examine the contribution of FA uptake via CD36 in the formation of HER2 + EMT-like CSCs, HCC1954, MDAMB361, EFM192A and HCC1569 cells were plated and grown in a 6-well plate at a density of 150,000–400,000 cells/well (according to the cell line) and incubated in culture media supplemented with FBS depleted (FBS-FA-) or not (FBS-FA +) of FA for 24 h, 48 h and 72 h. To evaluate the effects of selective WNT inhibition, HCC1954, MDAMB361, EFM192A and HCC1569 cells were treated with the WNT inhibitor LGK974 (Selleckchem, Karl-Schmid-Str.14, Munich, DE) as a single agent at final concentrations of 10 μM and 20 μM (HCC1954, MDAMB361 and EFM192A cells) or 15 μM and 30 μM (HCC1569 cells) or with 0.1% DMSO (Sigma‒Aldrich) as an internal control for 24 h and 48 h at 37 °C with 5% CO 2 . .. In parallel, to evaluate the effects of selective WNT activation, HCC1954, MDAMB361 and EFM192A cells were treated with the specific Isolation:Article Title: Role of the β-Catenin/REG Iα Axis in the Proliferation of Sessile Serrated Adenoma/Polyps Associated with Fusobacterium nucleatum Article Snippet: Human intestinal epithelial cell line Caco2 was obtained from ATCC (Manassas, VA, USA) and cultured in RPMI 1640 medium (Invitrogen, Carlsbad, CA, USA) with 10% fetal bovine serum (Biowest, Nuaillé, France) in a humidified incubator at 37 °C with an atmosphere of 5% CO2. .. The cells were treated with Reverse Transcription:Article Title: Role of the β-Catenin/REG Iα Axis in the Proliferation of Sessile Serrated Adenoma/Polyps Associated with Fusobacterium nucleatum Article Snippet: Human intestinal epithelial cell line Caco2 was obtained from ATCC (Manassas, VA, USA) and cultured in RPMI 1640 medium (Invitrogen, Carlsbad, CA, USA) with 10% fetal bovine serum (Biowest, Nuaillé, France) in a humidified incubator at 37 °C with an atmosphere of 5% CO2. .. The cells were treated with |
